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Under some pathophysiological circumstances, endothelium-derived vasoconstricting factors, such as angiotensin-1 and -II, vasoconstrictor prostaglandins and thromboxane A2, urotensin II, and superoxide anions, can be released and contribute to the paradoxical vasoconstrictor effects.[34] As was described, NO has an important role in vascular tone
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Small trials run in a single region, funded or supported by the maker, tend to report larger effects than big independent ones, partly because small studies that happen to land a positive result are the ones that get published while the null ones quietly disappear, and partly because looser blinding and outcome-grading let optimism leak into the numbers
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